SUMMARY: In my previous posts, I discussed HIV genes present in Sars-CoV-2, the virus that causes COVID-19, and showed how using HIV genes in recombinant viruses was the bread and butter of modern virology.
This post will introduce new evidence (published two days ago) of HIV/Sars mutant coronavirus chimeras discussed in NIH-funded research projects that led to the COVID pandemic. The research project discussed in this post was funded by NIH grant 1R01AI110964 and was titled “Understanding the Risk of Bat Coronavirus Emergence.” Grant 1R01AI110964 was given for research conducted by EcoHealth Alliance in cooperation with Dr. Ralph Baric of UNC and the Wuhan Institute of Virology.
In the last section, I will answer the question, “Can we trust science?”
Two days ago, 554 pages of previously secret documents related to certain NIH grants to the EcoHealth Alliance were released under FOIA to Judicial Watch.
The monetary grant, dispensing $3,748,715, funded the work of EcoHealth Alliance, Wuhan Institute of Virology, and Ralph Baric lab, towards the following (page 16):
- Assess CoV spillover potential
- Develop predictive models of bat CoV emergence risk and host range
- Test predictions of CoV inter-species transmission
Take a look at page 127:
The above page describes “receptor mutants and pseudoviruses,” specifically a mutant of HIV backbone with SARS-like “spike proteins.” These NIH-funded researchers created these HIV-SARS mutants to see how well they would infect human lungs and experimented on “humanized mice” whose lung cells resembled human cells.
These mutant viruses are, in a way, the opposite of Sars-Cov-2. They represent an HIV backbone with SARS-like spike protein.
Sars-Cov-2, on the contrary, is a coronavirus backbone with spike-protein including HIV genes. (Such a chimera is described in the DEFUSE proposal submitted by Peter Daszak to another government agency)
And yet, despite the dissimilarity, these mutants show how prevalent the lab work is that involves combining HIV and coronavirus genomes.
HIV is an exceptionally cunning virus: it integrates into the human DNA after infection. Such integration is called “reverse transcription,” whereby RNA from the virus becomes part of the DNA of human cells. HIV essentially edits the human genome and inserts its code into it. After that, human cells produce new HIV viral particles, as this NIH page explains.
However, HIV is not very contagious. It cannot target cellular receptors found in human lungs, for example. One cannot get HIV via the airborne route: HIV infections require blood-to-blood transmission, and HIV-carrying aerosols cannot infect human respiratory systems.
A pseudovirus that can infect human ACE2 cells, which are present in our lungs, is one step closer to airborne transmission: an aerosol carrying ACE-2-binding HIV mutant could infect someone upon being breathed in.
Is the research that creates ACE-2-infecting HIV mutants, which may be contagious via the respiratory route, safe? What if these recombinants escape the laboratory?
That does not seem safe to me!
And yet, in search of grant money, fame, and discoveries, virologists funded by the NIH conducted such experiments involving ACE2-infecting HIV chimeras with minimal oversight.
Their work gave us Sars-CoV-2 and the Covid pandemic. Again, Sars-Cov-2, a recombinant chimera carrying HIV genes on a coronavirus backbone, is NOT the same as the pseudovirus described above; it is something else but related.
The above shows that work on HIV/SARS coronavirus recombinants, funded by the NIH, was conducted by the same people whose cooperation with the Wuhan Institute of Virology gave us the Covid pandemic.
Almost every human was infected with the HIV-gene-carrying COVID virus. Every COVID-vaccinated human was injected with spike-protein-producing mRNA, which encoded the same HIV genes. We are now living through the consequences, with excess mortality continuing among the vaccinated countries.
